Is there anything that can prevent or delay the onset of Alzheimer’s disease?
Professor Ali believes that there is, and outlines the causes of brain toxicity and the role of oxystatic therapy.
Alzheimer’s disease is first and foremost an oxygen problem of the brain cells caused by the trio of toxicities of foods, the environment and thoughts, such as overload with heavy metals such as mercury and lead, toxic protein deposition, gut fermentation, and other factors that block oxygen’s energetic and detergent functions. In my book Oxygen and Aging (2000) I devoted a chapter to Alzheimer’s disease and the use of oxystatic therapies to prevent it and arrest its course in the early and intermediate stages.
OBSERVATIONAL STUDIES
Give me a group of 50 patients with Alzheimer’s disease, 25 in the early stage and 25 in the intermediate stage. I will use my Alzheimer’s Nutrient Protocol for a period of 18 weeks in a clinical observational study. The family members or care providers will be asked to complete an outcome questionnaire. I predict the following rates of positive responses in mental functions: early subgroup, over 50%; intermediate subgroup, between 25% and 50% (patients with advanced with Alzheimer’s disease do not respond to my protocol in a clearly demonstrable way).
While all the causes of brain toxicity cannot be addressed in just 18 weeks, evidence has been found to prevent or delay this devastating disease. The three-phase clinical outcome study described above is based on my experience or observational study.
NEW EVIDENCE
The purpose of my Alzheimer’s Protocol is to restore neuronal oxygen homeostasis. This subject assumes intense importance in the light of recent reports of compelling evidence that two types of tests, positron emission tomography (PET) scans of Alzheimer’s plaque in the brain and tests of spinal fluid, can predict the development of disease with near certainty years before the disease is clinically recognised.
DR ALI’S ALZHEIMER’S PROTOCOL
Phase I, 6 weeks
A. Intramuscular injections. Glutathione 400 mg, vitamin B12 5 000 mcg, vitamin B complex 2 ml, magnesium sulphate 500 mg.
The above protocol will be administered daily for 3 days, then on alternate days for 12 days, and finally on every third day for the remaining days of Phase I.
B. Intravenous infusions. My intravenous infusion protocol includes 400 mg of glutathione, 2 000 mg of magnesium sulfate, 500 mg of taurine, 5 000 mcg of vitamin B12, and multivitamins. It is administered once a week for 6 weeks. At the end of the first 6 weeks, family members or care providers are asked to complete a clinical outcome questionnaire.
Phase II, 6 weeks
All nutrient therapies will be discontinued for 6 weeks, after which the same family members or care providers are asked to complete the clinical outcome questionnaire.
Phase III, 6 weeks
Nutrient therapies as described in Phase I are reinstituted for the last 6-week period. Then the same family members or care providers are asked to complete the clinical outcome questionnaire.
In clinical practice, of course, I address all other oxygen issues and reduce the dose and frequency of injectable nutrient therapies in the long-term treatment plan.
Editor's note: You may find these articles helpful Feeding the Brain and Slowing its Ageing and The Dynamic Duo for Dementia Prevention.
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